Historical relation between Islam, Christianity and Judaism Essay

Historical relation between Islam, Christianity and Judaism - Essay Example Religion helps to provide a driving force for every man’s existence on Earth – it is a belief of faith that people indulge in, in order to feel secure and protected in terms of their actions with relation to others around them. Islam, Christianity and Judaism are three of the world’s oldest religions, perhaps stemming and growing from each other initially as per historical evidences; however, today, the three have created a niche for themselves in the world with great followings of their own. It is said that Judaism was the backing that gave way to Christianity and Islam and that is why most of their teachings and beliefs are very much similar. The three religions share a stark relationship with each other in terms of their history with a connection between their messengers, texts and teachings. If checked properly, one can also notice the similarities between their rites and rituals, and how it is only different names and the use of different languages that sepa rates the three from each other. The commonality between the three is that each religion believes in its own God, and has put major emphasis on the higher supreme power that rules the faith. It is said that these Gods were the creators of heaven and hell, and of man’s existence on Earth, and that is why each religion has its own set of beliefs and laws for every man to dedicate his life to serving God.. Traditionally speaking, all three religions are bound by history because of their connection with Prophets like Abraham, Moses, Jesus and Muhammad. They each were the servants of their Gods, and came as messengers to people in order to reveal the truth about life that God imparted to them. Abraham gave his people the message provided to him by God, Moses provided the Israelis with the Ten Commandments, and Jesus was the harbinger of the teachings of God and Hebrew prophets. Finally, Muhammad is said to have combined the above messages of God and reject any kind of idolatry whi le simply establishing some required laws for the rest of humanity to follow. According to history, it is said that Judaism formed the roots of religion from which Islam and Christianity grew with the help of Messiahs. All three religions had their own Messiahs that delivered messages of truth and peace to the rest of their followers. While taking the literal meaning of the word ‘God’ into account, all three religions date back to their creations; The Hebrew word for God is ‘Elohim’ which in Arabic or Islam can be translated as, and means, ‘Allah’. Jews were said to come from Isaac, the son of Abraham, and the Arabs from Ishmael, another son of Abraham. According to this theory, all three religions were born out of the brainchild of a single God, despite having branched out into separate entities. Thus, all of mankind is a child of Abraham. On reading the holy books pertaining to each religion, i.e., the Holy Bible or the Qur’an, many hi storians as well as people specializing in theology have pointed out that the texts and readings of all religions contain the same teachings in different languages. The names of the Prophets are also strikingly similar, with a few changes in the pronunciation due to being spoken about and written in different languages and scripts. With respect to history, taken from a pragmatic point of view, there is only one God and his sons were the ones to have brought his message to the rest of mankind, and this was done by three different elements, thus leading to the creation of different religions. Judaism, Christianity and Islam, gradually developed into powerful tools used for the purpose of politics and monarchy in different countries, among different people. Preaching to the people became a widely known concept in the early centuries of the world, and more and more people began to take to power just in order to be able to gather mass following. This came as a threat to the identity and existence of the religions as the purpose of existence for

Country Lovers Essay Example for Free

Country Lovers Essay A story of forbidden love on a South African farm, one child nothing more than a farm worker, as the other one prepares for boarding school. Both ignorant to the color of their skins, all they knew they were best friends playing together until they grew up and then it was not allowed. But when Paulus turns fifteen and goes to school things start to change he begins to realize the difference between boys and girls, and then the consequences behind his actions, but then it’s too late to turn back. What drew me into this story was the title country lovers, and then the forbidden love on a South African farm I wanted to see what it was about, curios to know if it was about two people of different races falling in love, creating racial relationships in a place that forbid it. And that was exactly what it was, two kids playing together as kids, a boy white and girl black, everyday playing on his father’s farm. What I couldn’t understand was why they were able to play as kids but not be able to socialize when they got older. If it was because of racial boundaries, why not keep them apart even when they are kids, then it wouldn’t be so confusing. The literary term and concept that best describes they way I am feeling about this would be Imagination because it expresses how the author was feeling about racial differences and allows the reader to feel what the author is writing about. I am using the Reader – Response approach to analyze my story because it asks you to connect with the literature, and find a personal link or imaginative entry into a story. And that is what this author makes you do. While reading this story I had a lot of different emotions like why did he change when he went to school, but when he came home he was with thebedi. Even though he met people at school whose family’s was prosperous famer’s? He still was bringing gifts to thebedi, she making gifts for him and both lying about where they came from and why. They had been sneaking around for months, so when she was eighteen and the farmer’s son was nineteen he left for veterinary college, Njabulo’s parents asked Thebedi’s parents to marry their son and they agreed on it, not telling Paulus or Njabulo that she might be pregnant. When the baby was born looked nothing like Njabulo, but he still was going to take care of her as if she was his own. This would be Satire because he should feel anger but instead he is willing to take care of her. My thinking didn’t change because in that time that’s what happens when two different races mix and have children, I just thank god it’s not like that now to the extreme it was then. Because I love people for who they are not because of where they come from. Because of the author’s background growing up in South Africa I can understand why she feels this way, they have different traditions and beliefs. This was a very good story; I enjoyed reading it and feeling what they were going through.

Essay --

The Black woman and her unique life experience have long since been neglected by mainstream media, historical accounts of events, and law makers just to name a few. For example, David Baker’s â€Å"Female Lynchings in the United States: Amending the Historical Record† emphasizes the carelessness and, as a result, inaccuracies of accounts taken of female lynchings from the early 1800s to the mid 1900s. The discrepancies, disorder, and missing information in the accounts Baker pulled from were all reminders of the frequency in which disrespect and disregard for Black Women and their experiences occurred. In modern day America, the Black woman’s experience is still lacking in the media. A prime example of the lack of presence of Black Women’s issues in the media is in Michelle Alexander’s â€Å"The New Jim Crow: Mass Incarceration in the Age of Colorblindness.† The book discusses mass incarceration as the new way in which Blacks are systematical ly controlled in America. Alexander makes a convincing argument that through colorblind racism, the criminal justice system as a whole strives to methodically control Black men. What Alexander, like so many other authors of critical race theory, fails to mention, is the ways in which Black women are systematically oppressed throughout the criminal justice system. Along with bearing the brunt of stigmatization, Black incarcerated women also often find themselves facing health problems that stem from life circumstances—some that also are racially influenced—outside prison-life, but are magnified once they are incarcerated. The reason Black incarcerated women’s health becomes more pressing during this period, other than the fact that they do not have their freedom, is that there is a large flaw in the way... ...s incarceration, this is referred to as the "conspiracy of silence" (Miller, pg. 133). The child may not want to openly discuss where their mother has gone. The child may also evoke develop negative social traits because of their inability to talk about their incarcerated mothers, which can potentially widen the gap between them and their incarcerated mother. We can all recognize the importance of children being able to spend a certain amount of time with their mothers, but in prison that time is often cut short or non-existent which hinders the relationship between the mother and child. Some states are unwilling to provide children's visitation at their correctional facilities, and as a result the child becomes a victim of the criminal process. The child is paying for the crime that their mother committed by not being able to spend enough quality time with them. Essay -- The Black woman and her unique life experience have long since been neglected by mainstream media, historical accounts of events, and law makers just to name a few. For example, David Baker’s â€Å"Female Lynchings in the United States: Amending the Historical Record† emphasizes the carelessness and, as a result, inaccuracies of accounts taken of female lynchings from the early 1800s to the mid 1900s. The discrepancies, disorder, and missing information in the accounts Baker pulled from were all reminders of the frequency in which disrespect and disregard for Black Women and their experiences occurred. In modern day America, the Black woman’s experience is still lacking in the media. A prime example of the lack of presence of Black Women’s issues in the media is in Michelle Alexander’s â€Å"The New Jim Crow: Mass Incarceration in the Age of Colorblindness.† The book discusses mass incarceration as the new way in which Blacks are systematical ly controlled in America. Alexander makes a convincing argument that through colorblind racism, the criminal justice system as a whole strives to methodically control Black men. What Alexander, like so many other authors of critical race theory, fails to mention, is the ways in which Black women are systematically oppressed throughout the criminal justice system. Along with bearing the brunt of stigmatization, Black incarcerated women also often find themselves facing health problems that stem from life circumstances—some that also are racially influenced—outside prison-life, but are magnified once they are incarcerated. The reason Black incarcerated women’s health becomes more pressing during this period, other than the fact that they do not have their freedom, is that there is a large flaw in the way... ...s incarceration, this is referred to as the "conspiracy of silence" (Miller, pg. 133). The child may not want to openly discuss where their mother has gone. The child may also evoke develop negative social traits because of their inability to talk about their incarcerated mothers, which can potentially widen the gap between them and their incarcerated mother. We can all recognize the importance of children being able to spend a certain amount of time with their mothers, but in prison that time is often cut short or non-existent which hinders the relationship between the mother and child. Some states are unwilling to provide children's visitation at their correctional facilities, and as a result the child becomes a victim of the criminal process. The child is paying for the crime that their mother committed by not being able to spend enough quality time with them.

Mghf

Huntington’s disease is an autosomal dominant (Harper et al, 1991) neurodegenerative disorder characterized by involuntary movements, cognitive loss, and psychiatric problems as described by Martin and Gusella (1986). These symptoms are related to the death of medium spiny projection neurons in the caudate nucleus, putamen, and the cortex (Reiner et al, 1988). In later stages of the disease, even areas such as the hippocampus and hypothalamus gets affected as well (Kassubek et al, 2004). Huntington’s disease has a prevalence rate of approximately 1 in 10,000 Caucasian populations (Harper et al, 1991).Patients with Huntington’s disease most typically display a choreic movement disorder involving involuntary writhing movements observed by George Huntington himself. The age of onset of Huntington’s disease seem to be normally distributed around the average age of 35 to 42, with small number of cases developing in patients younger than 20 years of age or older than 60 years of age (Andrew et al, 1993). The majority of juvenile patients, whose disease onset are at age 20 years or less, have inherited the paternally defected gene (Andrew et al, 1993).Patients usually died within 15 to 20 years of disease onset. Gusella et al (1983) first determined the location of the genetic mutation in Huntington’s disease to be the short arm of chromosome 4. It was after another 10 years did the researchers of The Huntington’s Disease Collaborative Research Group (1993) able to discover that a gene in chromosome 4 containing a trinucleotide repeat of CAG was abnormally expanded in diseased individuals. The IT15 (interesting transcript 15) gene, dubbed the huntingtin gene by the group, codes for the huntingtin gene.This CAG repeat, which translates into a poly-glutamine stretch, is highly polymorphic among the population ranging from 11 to 34 copies on normal individuals. However, in individuals with Huntington’s disease it expanded to more than 42 repeats and increasing to upwards of 100 (The Huntington’s Disease Collaborative Research Group, 1993). This provides evidence showing that the mutant huntingtin protein seems to be toxic to its native cells and confers a disease state to individuals with an extended length of repeats. There is also a correlation between the CAG length and disease onset as shown by Andrew et al (1993).The mutated elongated huntingtin protein is cut by enzymes into fragments and the fragments begin to form abnormal clusters, neuronal intranuclear inclusions (NIIs), inside cells. These clusters can also act to recruit normal proteins to adhere together as well (Davies et al, 1997). This was originally thought to cause the pathogenesis of Huntington’s disease. However, more recent studies have shown that the presence of NIIs is actually a coping response to the toxicity of mutant huntingtin proteins and acts to prolong the life of the cells and reduce intracellular mutant huntingtin in neighbouring neurons (Arrasate et al, 2004).The exact function of the wild-type huntingtin protein are unclear, however many efforts have been made in understanding its native functions. Nasir et al (1995) showed that homozygous huntingtin homologs in mice died before embryogenesis could occur and that heterozygotes displayed similar deficits as diseased human patients. Wild-type huntingtin is also crucial for establishing and maintaining neuronal identity, especially in cortex and striatum (Reiner et al, 2001).Current data can provide the conclusion that normal huntingtin protein has actions important for development in mammals. In vitro, wild-type huntingtin have been shown to act to protect brain cells from apoptotic stimuli, such as serum deprivation, mitochondrial toxins, or the transfection of death genes (Cattaneo et al, 2005). Wild-type huntingtin protein, not mutated, stimulates brain-derived neurotrophic factor (BDNF) production by acting at level of Bdnf tra nscription. BDNF is very important for survival of striatal neurons (Cattaneo et al, 2005).Intracellularly, huntingtin protein has been found to associate with various organelles such as the nucleus, endoplasmic reticulum, and Golgi complex (Cattaneo et al, 2005). It has also been found in neurites and at synapses, where it associates with vesicular structures and microtubules (Li et al, 2003). This characteristic has been shown to enhance vesicular transport of BDNF along microtubules (Gauthier et al, 2004). On a similar note, huntingtin interacts with a number of cytoskeletal and synaptic vesicle proteins that are essential for exo- and endocytosis at synaptic terminals.Wild-type huntingtin binds directly to the Src homology 3 domain of postsynaptic density protein 95, which binds NMDA and kainite receptors. This activity is decreased in mutant proteins and can lead to overactivation or sensitization of NMDA receptors (Cattaneo et al, 2005). Aside from the toxicity of the mutated huntingtin protein, the loss of normal huntingtin protein also seems to add to the pathogenesis of Huntington’s disease. Presence of only mutant huntingtin protein results in massive apoptotic cell death in the testes of male mice (Leavitt, 2001).However, no apoptosis can be seen in testes of mice expressing human mutant huntingtin when wild-type huntingtin is expressed as well (Leavitt, 2001). It was also seen that in mice, the absence of wild-type huntingtin protein led to a worsening of striatal atrophy and neuronal loss, and a significant decrease in neuronal cross-sectional area compared to mice that had wild-type huntingtin present (Cattaneo et al, 2005). Huntington’s disease still remains incurable to this day. However, many treatments are available for treatments of its symptoms.Chorea, the hallmark of the disease is a major target for many treatments. Such drugs include dopamine-depleting agents, dopamine antagonists, benzodiazepines, glutamate antagonists, ac etylcholinesterase inhibitors, dopamine agonists, antiseizure medications, cannabinoids, lithium, deep brain stimulation and fetal cell transplantation (Frank and Jankovic, 2010). One notable drug currently in use is Tetrabenazine, which is the only US FDA-approved drug for treatment of Huntington’s disease (Frank and Jankovic, 2010).The drug acts by reversibly inhibiting the central vesicular monoamine transporter type 2, this cause a depletion of dopamine (Bagchi, 1983). The main area of effect for tetrabenazine is in the caudate nucleus, putamen, and the nucleus accumbens, all areas known to be responsible for the major pathology of the disease. The Huntington Study Group (2006) was able to demonstrate the efficacy of tetrabenazine in a double-blind, placebo-controlled trial. Subjects who received tetrabenazine showed a change from the baseline in the maximal chorea score of the UHDRS.Compared to the baseline, treatment resulted in a reduction of 5. 0 units in chorea compa red with a 1. 5 unit reduction for the placebo group. A second symptom targeted for treatment to allow patients to function normally is the psychiatric problems. Dopamine receptor blocking agents are commonly used as anti-psychotics in order to treat psychosis associated with Huntington’s disease (Frank and Jankovic, 2010). There are many ethical issues that have been associated with Huntington’s disease patients, in particular pertaining to the application of genetic testing.The expanded CAG repeats associated with Huntington’s disease (Gusella et al, 1983) are used as a predictive testing to determine the risk of a person for developing inherited HD gene. The debate of ethics with respect to genetic testing for Huntington’s disease seems to lie on several major factors including: autonomy, beneficence, confidentiality, and justice (Huggins et al, 1990). The question remains whether or not the current use of genetic predictive testing is ethical. Autonom y refers to the respect for the individual’s right to make an informed decision about an action that may have a profound effect on his or her life.The patients should feel no pressure from physicians or institutions with regards to their decision for genetic testing. Also, they should also have a full understanding of the consequences and implications of their decisions. This would require physicians to provide all the necessary information to the patients so that they are informed about the testing. If a woman refuses to get tests done for her baby even though her relatives are with her insisting on getting it performed, the physician cannot allow the testing to be performed.Beneficience is summarized by the phrase â€Å"first do no harm† (Huggins et al, 1990). This is an important factor when the results of the test may reveal genetic information about oneself. This implies not only avoiding harm to patient but also preventing harm to other individuals, which may inc lude family members of patients. Due to the genetic characteristics of Huntington’s disease, family members can be both directly and indirection influenced by the results of the test.If for example a pregnant woman decides to get an ultra sound for her baby in order to find out if the baby has spina bifida, and upon confirmation by the physician decides that she wants to give birth at home as opposed to in a hospital where the baby could be treated correctly in order to minimize the risk of the disease. The physician should take any action he can in order to avoid any risk to the health of the baby. Confidentiality with genetic tests applies the same as with any other form of personal information. It should not be disclosed to any other third parties, which includes family members as well.For example, if two siblings were to go and receive a CAG repeat test at the same time and one sibling asks the physician about the results of the other sibling. The physician cannot disclos e any information or they will violate the confidentiality of the patient. Justice simply means equal access to health services and information to all. This also includes long-term support and guidelines for testing as a medical service so that the service is not difficult to obtain if needed. For example, even though Cystic Fibrosis is mostly only prominent in Caucasians, the testing should still be offered to regnant women of other ethnicities as well. Other ethical dilemmas still exist with current genetic testing technologies. One particularly pertaining to Huntington’s disease is whether or not the information is obtained even though there is no cure for the disease. In diseases such as phenylketonuria (PKU), after newborn screening, dietary interventions will allow the individuals with the condition to lead healthy and normal lives (Lea et al, 2005). However, for Huntington’s disease, even after knowing that the disease will affect the individual what can be done ?Therefore, would it be better to not know at all? By testing and finding out that an individual has the mutation that will eventually lead to the development of Huntington’s disease, the individual can attend to the initial development of symptoms and as a result may be treated much earlier. This can result in a more favourable prognosis. Similarly, the individual can live more cautiously as to prevent further exacerbation of their condition. On a more subjective level, the individual may use the knowledge to live a more fulfilling life knowing that he or she may not live as long as the rest of the population.Conversely, the information can also be used negatively. There can be a lot of discrimination in terms of applying for employment and health insurance. This information would also impact the ambition and life goals of the individual, the amount of psychological harm that comes with eventually developing an incurable and lethal neurodegenerative disease is extremely larg e. Similarly, would they still pursue to have a family and children if they knew that they had a very high risk of dying at a young age? This particular issue should be resolved by following the ethical factors listed before.As long as the individual is informed, are being prevented from harm, information not shared with any third party members, and has access to healthcare services, then being tested for Huntington’s disease will be ethically correct. This knowledge will give them insight on how to live the rest of their life. Another dilemma that has Harper, P. S. , Morris, M. J. , Quarrell, O. , Shaw, D. J. , Tyler, A. , and Youngman, S. (1991). The epidemiology of Huntington’s disease. J. Med. Genet. 89: 365-376. Martin, J. B. , and Gusella, J. K. (1986).Huntington’s disease: pathogenesis and management. N. Engl. J. Med. 315: 1267-1276. Andrew, S. E. , Goldberg, Y. P. , Kremer, B. , Telenius, H. , Theilmann, J. , Adamn, S. , Starr, E. , Squitieri, F. , Lin, B. , Kalchman, M. A. , Graham, R. K. , and Hayden, M. R. (1993). The relationship between trinucleotide (CAG) repeat length and clinical features of Huntington’s disease. Nat Genet. 4: 398-403. The Huntington’s Disease Collaborative Research Group (1993). A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes.Cell. 72: 971-983. Gusella, J. F. , Wexler, N. S. , Coneally, P. M. , Naylor, S. L. , Anderson, M. A. , Tanzi, R. E. , Watkins, P. C. , Ottina, K. , Wallace, M. R. , Sakaguchi, Y. A. , et al. (1983). A polymorphic DNA marker genetically linked to Huntington’s disease. Nature. 306: 234-238. Reiner, A. , Albin, R. L. , Anderson, K. D. , D’Amato, C. J. , Penney, J. B. , and Young, A. B. (1988). Differential loss of striatal projection neurons in Huntington disease. Proc. Natl Acad. Sci. USA. 85: 5733–5737. Kassubek, J. , Gaus, W. , and Landwehrmeyer, G. B. (2004).Evidence for more w idespread cerebral pathology in early HD: and MRI-based morphometric analysis. Neurology. 62: 523-524. Nasir, J. , Floresco, S. B. , O’Kusky, J. R. , Diewert, V. M. , Richman, J. M. , Zeisler, J. , Borowski, A. , Marth, J. D. , Philips, A. G. , and Hayden, M. R. (1995). Targeted disruption of the Huntington’s disease gene results in embryonic lethality and behavioral and morphological changes in heterozygotes. Cell. 81: 811-823. Cattaneo, E. , Zuccato, C. , and Tartari, M. (2005). Normal huntingtin function: alternative approach to Huntington’s disease.Nat Rev Neurosci. 6: 919-930. Li, J. Y. , Plomann, M. & Brundin, P. (2003). Huntington’s disease: a synaptopathy? Trends Mol. Med. 9: 414–420. Reiner, A. , Del Mar, N. , Meade, C. A. , Yang, H. , Dragatsis, I. , Zeitlin, S. , and Goldowitz, D. (2001). Neurons lacking huntingtin differentially colonize brain and survive in chimeric mice. J. Neurosci. 21: 7608-7619. Gauthier, L. R. , Charrin, B. C. , B orrell-Pages, M. , Dompierre, J. P. , Rangone, H. , Cordelieres, F. P. , De Mey, J. , MacDonald, M. E. , Lessmann, V. , Humbert, S. , and Saudou, F. 2004). Huntingtin controls neurotrophic support and survival of neurons by enhancing BDNF vesicular transport along microtubules. Cell. 118: 127-138. Leavitt, B. R. , Guttman, J. A. , Hodgson, J. G. , Kimel, G. H. , Singaraja, R. , Vogl, A. W. , and Hayden, M. R. (2001). Wild-type hungtingtin reduces the cellular toxicity of mutant huntingtin in vivo. Am. J. Hum. Genet. 68: 313-324. Davies, S. W. , Turmaine, M. , Cozens, B. A. , DiFiglia, M. , Sharp, A. H. , Ross, C. A. , Scherzinger, E. , Wanker, E. E. , Mangiarini, L. , and Bates, G. P. (1997).Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation. Cell. 90: 537-548. Arrasate, M. , Mitra, S. , Schweitzer, E. S. , Segal, M. R. , and Finkbeiner, S. (2004). Inclusion body formation reduces levels of mutant huntingtin an d the risk of neuronal death. Nature. 431: 805-810. Frank, S. , and Jankovic, J. (2010). Advances in the pharmacological management of Huntington’s disease. Drugs. 70: 561-571. Bagchi, S. P. (1983). Differential interactions of phencyclidine with tetrabenazine and reserpine affecting intraneuronal dopamine.Biochem Pharmacol. 32: 2851-6 Huntington Study Group. (2006). Tetrabenazine as antichorea therapy in Huntington disease: a randomized controlled trial. Neurology. 66: 366-72 Huggins, M. , Bloch, M. , Kanani, S. , Quarrell, O. W. , Theilman, J. , Hedrick, A. , Dickens, B. , Lynch, A. , and Hayden, M. (1990). Ethical and legal dilemmas arising during predictive testing for adult-onset disease: the experience of Huntington disease. Am. J. Hum. Genet. 47: 4-12. Lea, D. H. , Williams, J. , and Donahue, M. P. (2005). Ethical issues in genetic testing. J. Midwifery Womens Health. 550:234-240.

Drama assessment on dennis kellys ‘DNA’

Lea being the main narrator, sat up on her knees, Inviting the audience In to listen and seemed more comforting and engaged, whereas Phil communicated with the audience with the lack of focus and response, showing have AR away Phil really is to understanding Leash's thoughts and feelings. The actors facial features were kept very minimal, neural expressions which showed no emotion allowing the other two character to really communicate and show the audience what they really think and feel.We did this to support our Interpretation of the characters and what we got from the play which was that Lea had deep feelings for Phil, which he took for granted. I played Leash's conscience, alongside†¦.. , who played Phial's. I and †¦ Walked up to each other hesitantly, showing the lack of communication between the characters as their unsure of their relationship. We help our hands out towards each other after we had separated to communicate through the use of gesture, to the audience th at we do truly need each other, we just can't show it.This was my idea, and I think it was successful because I really wanted to show to the audience the true feelings of the two characters and I think this was a good moment to communicate with the audience. I contrasted with tone of voice, as she uses a soft yet pleading tone, I respond to it with an aggressive tug of Cam's arm, silently beginning him to respond. Leash's self-conscious, in coherent character and Phial's stubborn blindness, tops them communicating, and I believe this is why their relationship is so strained and unclear. Ã'›â€ ¦ Asked us to enter the stage one by one, and to create a still image of a character, showing them at the start and end of the play. We had to then bring our image to life, using role play to show what we would have thought at those points. I was playing Brian, a miss interpreted character, which didn't get enough attention even though the situation was clearly affecting him in a strong ph ysiological level. For my first still image I stood with a slouch, protectively wrapping my arms around my odd, by lowering onto a smaller level, symbolizes Briar's vulnerability and scared nature.Avoiding eye contact too communicate with the audience, showing how uncomfortable Brain was, repeating â€Å"l can't go In there† and â€Å"l feel guilty, stuttering my words. By slurring and stuttering the words shows the audience that I'm not an overly confident character and the pressure and guilt Is getting too much for me, communicating my feelings and status within the group, saying things Like â€Å"they can't make me go in their again†, showing that the rest of the group takes advantage of his weak nature, which Is showed throughout the play. On the other hand, when †¦. Wowed his Interpretation, he showed Brian forcefully splitting out his words, showing his distaste towards the other characters In the play, glaring at the audience, communicating via facial expr ession, whereas I relied on my body language. After delivering my narration, I used a melting machine to change my Briar's over enthusiastic, UN naturalistic character in contrast to the serious situation he was in. At the end of the play, you're informed of Briar's diagnosed mental issue, so I tried to communicate that with over enthusiastic body moment and gestures, to express the erratic state of his mind.It was intriguing to see where the characters ended up after being involved in murder, and Briar's account has got to be the most shocking and legit, Dennis Kelly is clever with the way he shows his character. Miss asked us to get into larger groups to perform a conscious alley to show Phial's inner battle for his decision to kill Adam. My group was the most effective as we added various bits of narration in the mist of some role playing in unison. I was on the side which was trying to skillfully persuade Phil to choose the moral decision, too not killWe started off with the lig hts off, eliminating the audiences senses, all laughing in high pitches, moving around the blocks to make the audience vulnerable, communicating Adams feelings which he would of felt when he woke up with the woods surrounding him. I silenced the laughing and the darkness by saying the line â€Å"So however you been living? â€Å", a seemingly innocent, naturalistic question, which then .. Elaborates and turns the scene from Statisticians inspired to Retard in a quick change of pace, which is heightened with the rest of us hitting the block and our legs and add a form of an ritual.